Lab Report New
Lab Report New
I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:21PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 06:15PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
HAEMATOLOGY
Test Name Value Unit Bio Ref.Interval
Basophils 0 % <2
Method -Flowcytometry method using a semiconductor
Page 1 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:21PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 06:15PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
HAEMATOLOGY
Test Name Value Unit Bio Ref.Interval
Page 2 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 06:47PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 08:23PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
BIOCHEMISTRY
Test Name Value Unit Bio Ref.Interval
CLINICAL NOTE:-
Glucose is a primary cellular energy source. Fasting plasma glucose concentrations and tolerance to a dose of
glucose are used to establish the diagnosis of diabetes mellitus and disorders of carbohydrate metabolism.
Test performed on BC_AU5800
Page 3 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 09:47PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
BIOCHEMISTRY
Test Name Value Unit Bio Ref.Interval
Page 4 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 09:47PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
BIOCHEMISTRY
Test Name Value Unit Bio Ref.Interval
LIPID PROFILE
Cholesterol Total,Serum 164 mg/dL < 200
Method -CHOD - PAP
NATIONAL LIPID ASSOCIATION TOTAL CHOLESTEROL in TRIGLYCERIDE in LDL CHOLESTEROL in NON HDL CHOLESTEROL
RECOMMENDATIONS (NLA-2014) mg/dL mg/dL mg/dL in mg/dL
Optimal < 200 < 150 < 100 < 130
Above Optimal -- - --- 100 - 129 130 - 159
Borderline High 200 - 239 150 - 199 130 - 159 160 - 189
High > 240 200-499 160-189 190-219
Very High --- > 500 > 190 > 220
Note:-
1. Estimation of LDL cholesterol by Direct method is recommended when Triglycerides >400 mg/dL.
2. Measurements in the same patient can show physiological & analytical variations.
3. Three serial samples 1 week apart are recommended for Total Cholesterol, Triglycerides, HDL & LDL Cholesterol.
4. Certain conditions such as acute illness, stress, pregnancy, dietary changes especially changes in intake of saturated fatty acids,
lipid lowering drugs, alcohol or prednisone may cause variation in lipid levels.
5. As per NCEP guidelines, all adults above the age of 20 years should be screened for lipid status. Selective screening of
code for verification
Page 5 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 09:47PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
BIOCHEMISTRY
Test Name Value Unit Bio Ref.Interval
children above the age of 2 years with a family history of premature cardiovascular disease or those with at least one parent
with high total cholesterol is recommended.
6. It is recommends, for routine screening, a fasting lipid profile is not mandatory. Both fasting and non-fasting lipid profiles are
important for managing Indian patients with dyslipidemia. In most patients, there is usually a clinically unimportant increase in
TG concentrations 2-6 hours after eating normal meals. Non fasting levels are required to determine post prandial
hypertriglyceridemia which may be more significant predictor of CVD risk.
Page 6 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 07:23PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
BIOCHEMISTRY
Test Name Value Unit Bio Ref.Interval
Page 7 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 07:23PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
BIOCHEMISTRY
Test Name Value Unit Bio Ref.Interval
Iron Profile
Iron ,Serum 117 µg/dL 70 - 180
Method -TPTZ
UIBC(Unsaturated Iron Binding Capacity) 252 µg/dL 155 - 355
Method -Nitroso PSAP
TIBC(Total Iron Binding Capacity) 369.00 ug/dL 225.0 - 535.0
Method -Calculated
Comment:
Most body iron is found in hemoglobin. The serum measurement of iron is useful in the differential diagnosis of anemia,
iron deficiency anemia, thalassemia, possible sideroblastic anemia, and iron poisoning. Total iron-binding capacity in
serum, representing transferrin concentration in iron-binding capacity, is a useful index of nutritional iron status.
Iron deficiency anemia is characterized by a decreased serum Fe, increased TIBC or transferrin, and a decreased
transferrin saturation. Serum TIBC is increased in iron deficiency. Serum TIBC is decreased in anemia of chronic
disease.
INTERPRETATION:
Serum iron concentration is decreased in many but not all patients with iron deficiency anemia; Inflammatory disorders such as acute
infection, immunization, and myocardial infarction; acute or recent haemorrhage; malignancy; kwashiorkor; late pregnancy; menstruation
and nephrosis. Serum iron concentration diminishes markedly in patients who are beginning to respond to specific therapy for anemias for
other causes e.g. treatment nof pernicious anemia with Vit B12.Greater than normal concentrations of serum iron occur in iron over-loaded
disorder such as haemochromatosis and in acute iron poisoning following oral or parenteral iron administration. Iron level may also be
increased in acute hepatitis, lead poisoning, acute leukemia, thalassemia or oral contraception.
Test performed on BC_AU5800_2
Page 8 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 06:49PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
IMMUNOLOGY
Test Name Value Unit Bio Ref.Interval
THYROID PROFILE
T3 (Total Triiodothronine) , Serum 0.82 ng/ml 0.35 - 1.93
Method -CMIA
Note:
1.Total T3 & T4 levels measure the hormone which is in the bound form and is not available to most tissues.
2. Severe systemic illness affects the thyroid binding proteins and can falsely alter Total T4 levels in the absence of a primary thyroid
disease. Hence Free T3 & T4 levels are recommended for accurate assessment of thyroid dysfunction.
Note: TSH levels are subject to circadian variation, reaching peak levels between 2 - 4.a.m. and at a minimum between 6-10 pm The
variation is of the order of 50% .hence time of the day has influence on the measured serum TSH concentrations.
Clinical Use:
Diagnose Hypothyroidism and Hyperthyroidism
Monitor T4 replacement or T4 suppressive therapy
Quanitify TSH levels in the subnormal range
Increased Levels: Primary hypothyroidism, Subclinical hypothyroidism, TSH dependent, HyperthyroidismThyroid hormone resistance
Decreased Levels: Graves disease, Autonomous thyroid hormone secretion, TSH deficiency
Page 9 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 07:48PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
IMMUNOLOGY
Test Name Value Unit Bio Ref.Interval
Expected Values
Deficiency : < 20
Insufficiency : 20 - 30
Sufficiency : 30 - 100
Toxicity : > 100.0
Note: It should be taken into cosideration that differences in Vitamin D (25-OH) levels may exist with respect to gender, age, season, geographical
area, latitude and ethnic groups
Comments
Vitamin D Total assay is used as an aid in the assesment of Vitamin D sufficiency in adults.
Vitamin D is acquired either by exposure to sunlight or ingestion of food containing vitamin D. It is metabolized to vit D, 25 hydroxy in the liver in the
first step by vit D,25-hydroxylase system. A small amount of it further gets metabolized by hydroxylation in kidney to vit D 1,25 dihydroxy. Since vit D,
25 hydroxy is the predominant circulating form of Vit D in normal population, it is considered to be the most reliable index of vit D status.
Vitamin D is essential for bone health. In children, severe deficiency leads to bone-malformation, known as rickets. Milder degrees of insufficiecy
are believed to cause reduced efficiency in the utilization of dietary calcium.
The measurement of 25-OH-D is becoming increasingly important in the management of patients with various disorders of calcium metabolism
associated with Rickets, neonatal hypocalcemia, pregnancy, nutritional and renal osteodystrophy, hypoparathyroidism, and postmenopausal state.
Increased levels are found in Vit D intoxication.
Decreased levels are detected in Rickets, osteomalacia, secondary hyperparathyroidism, malabsorption of vit D (e.g. liver diseases, cholestasis),
and diseases that increase Vit D metabolism (viz. Tuberculosis, sarcoidosis, primary hyperparathyroidism).
Recent studies have associated vitamin D deficiency with increased risk for cancer, autoimmune disease, infectious disease, cardiovascular
disease, and many chronic diseases.
Chronic severe vitamin D deficiency in infants and children causes bone deformation commonly known as rickets, while in adults, proximal
muscle weakness, bone pain and osteomalacia may develop.
Page 10 of 11
Sample Collected At :LUDHIANA 4(Gurudwara Kuhisar Sahib, A.T.I Road, Ludhiana)
Name: Mr. TARANJEET SINGH UID.: 800542
Age/Gender: 50 Y/Male Registered on: 19/Oct/2024 09:31AM
Mobile: 9417507880 Sample collected on: 19/Oct/2024 09:48AM
Lab No: 0912410190021 Sample received on: 19/Oct/2024 05:30PM
BarcodeNo: 11331188 Report released on.: 19/Oct/2024 06:53PM
Referred By.: Self Report status: Final
Test Name: LIVER PROFILE (LFT),LIPID PROFILE,KIDNEY PROFILE (KFT),Iron Profile,Vitamin B12,THYROID
PROF
Net Amt: Rs.800 Paid Amt: Rs.800 Balance Amt: Rs.0
Life Care Foundation
IMMUNOLOGY
Test Name Value Unit Bio Ref.Interval
Vitamin B12
Vitamin B12 ,Serum 232 pg/mL 187 - 883
Method -CMIA
Comments :-
Vitamin B12 performs many important functions in the body, but the most significant function is to act as coenzyme for reducing ribonucleotides to
deoxyribonucleotides, a step in the formation of genes. Inadequate dietary intake is not the commonest cause for cobalamine deficiency. The most common cause is
malabsorption either due to atrophy of gastric mucosa or diseases of terminal ileum. Cobalamine deficiency leads to Megaloblastic anemia and demyelination of large
nerve fibres of spinal cord. Normal body stores are sufficient to last for 3-6 years. Sources of Vitamin B12 are liver, shellfish, fish, meat, eggs, milk, cheese & yogurt.
Decreased Levels :-
Lack of Intrinsic factor: Total or partial gastrectomy, Atrophic gastritis, Intrinsic factor antibodies
Malabsorption: Regional ileitis, resected bowel, Tropical Sprue, Celiac disease, pancreatic insufficiency, bacterial overgrowth & achlorhydria
Loss of ingested vitamin B12: fish tapeworm
Dietary deficiency: Vegetarians
Congenital disorders: Orotic aciduria & transcobalamine deficiency
Increased demand: Pregnancy specially last trimester
Increased Levels:-
Chronic renal failure, Congestive heart failure, Acute & Chronic Myeloid Leukemia, Polycythemia vera, Carcinomas with liver metastasis, Liver disease, Drug
induced cholestasis & Protein malnutrition.
Limitations - interference
The assay is unaffected by icterus (bilirubin ≤ 1112 μmol/L or ≤ 65 mg/dL),hemolysis (Hb ≤ 0.025 mmol/L or ≤ 0.04 g/dL), lipemia (Intralipid≤ 17.1 mmol/L or ≤ 1500 mg/dL), biotin (≤ 205
nmol/L or ≤ 50 ng/mL), IgG ≤ 28 g/L, IgA ≤ 16 g/L and IgM ≤ 10 g/L. Criterion: Recovery within ± 10 % of initial value with samples > 200 pg/mL and ≤ ± 20 pg/mL with samples ≤ 200
pg/mL. Samples should not be taken from patients receiving therapy with high biotin doses (i.e. > 5 mg/day) until at least 8 hours following the last biotin administration. No interference was
observed from rheumatoid factors up to a concentration of 1500 IU/mL. In vitro tests were performed on 16 commonly used pharmaceuticals. No interference with the assay was found. In rare cases,
interference due to extremely high titers of antibodies to analyte specific antibodies, streptavidin or ruthenium can occur. These effects are minimized by suitable test design. Because intrinsic factor is
typically used as the binding protein in serum vitamin B12 assays, anti-intrinsic factor antibodies (which are common in pernicious anemia) can lead to elevated vitamin B12 measurement
values.2,11,12 The Elecsys Vitamin B12 II assay is designed to avoid interference due to anti-intrinsic factor antibodies.13 For diagnostic purposes, the results should always be assessed in
conjunction with the patient's medical history, clinical examination and other findings.
Note: The presence of immunoglobulin-vitamin B12 complexes may cause unexpectedly high values of vitamin B12.14,15
Page 11 of 11