Group-1b Myocardial Infarction
Group-1b Myocardial Infarction
College of Nursing
A Case Study
Presented to the College of
NURSING
University of Perpetual Help System DALTA
Prepared by:
Group 1-B
IGPIT, KEN JOMAR
JAZUL, ANGELICA
REYES, JEAN BRIENNE
SORIMA, CAMILLE
SUMARCA, KRIS DIMPLE
TAVARES, JELSEY KAYE
Clinical Instructor:
Mrs. Linda Sobrevega RN, MAN, LPT
Page
TITLE PAGE . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
TABLE OF CONTENTS . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
INTRODUCTION . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
OBJECTIVES . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
CHIEF COMPLAINT . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
PATIENT PROFILE . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
PATHOPHYSIOLOGY . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
DRUG STUDY . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
REFERENCES . . . . . . . .. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .
INTRODUCTION
Myocardial Infarction is the process by which portions of myocardial cells in the heart
are irreversibly damaged. MI is caused by decreased blood flow in a coronary artery due to
angina. Other causes of an MI include vasospasm of a coronary artery; decreased oxygen supply;
and increased demand for oxygen. In each case, a profound imbalance exists between myocardial
by characteristic changes on the electrocardiography (ECG). One of those ECG changes is the
typical elevation in the “ST segment” which is called STEMI. On the contrary, the absence of
ST-segment elevation and the presence of positive cardiac biomarkers such as troponin are called
NSTEMI.
Some of the symptoms that can be seen in patients having myocardial infarction are
pressure or tightness in the chest, pain in the chest, back, jaw, and other areas of the upper body
that lasts more than a 20 minutes or that goes away and comes back and shortness of breath.
According to Jarayadj et al. (2018), The most common form of CHD is the myocardial
infarction. It is responsible for over 15% of mortality each year, among the vast majority of
people suffering from non-ST-segment elevation myocardial infarction (NSTEMI) than ST-
infarction (MI) every 60 seconds. The prevalence of myocardial infarction (MI) is higher in men
in all age-specific groups than women. In addition, the study estimated that globally, IHD affects
around 126 million individuals (1,655 per 100,000), which is approximately 1.72% of the world's
population. Nine million deaths were caused by IHD globally. Men were more commonly
affected than women, and incidence typically started in the fourth decade and increased with age.
STEMI has the highest rate of cardiovascular diseases with an average mortality rate of
10 percent based on the health facility records in the Philippines. More than 42,000 people were
admitted to hospitals due to acute myocardial or coronary syndrome, while 3,800 die every year
This study focuses on Mr. M.I, a 46-year-old man, admitted on October 25, 2021 at the
Perpetual Help Medical Center with chief complaints of DOB and nausea.
OBJECTIVES
General Objective:
The primary concern of this study is to provide knowledge and enhance essential skills in care of
patients with myocardial infarction.
Specific Objectives:
1. To know and have a clear understanding of the factors that can lead to myocardial
infarction.
2. To establish rapport with the patient and significant others for an effective nurse-patient
interaction.
CHIEF COMPLAINT
Height 5’9”
Weight 93 kg
Birthdate: 01/01/1975
Nationality: Filipino
HEMATOLOGY DEPARTMENT
Red Blood Cell 4.26 4.50-5.50 x Patient with MI commonly develop anemia
10^12/L that causes decreased oxygen supply.
· Reference: Zafari et al., (2019).
(L) “Myocardial Infarction”
HS Troponin T 512 ng/ml Less than The heart releases troponin into the blood
0.40 following an injury, such as a heart attack.
Very high troponin levels usually mean that
a person has recently had a heart attack.
X-RAY SECTION
EXAMINATION: CHEST PORTABLE
FINDINGS:
- Pulmonary vascularity is increased with hilar fuzziness.
- Right hemidiaphragm and costophrenic sulcus are obscured by the left cardiac border.
IMPRESSION:
- Pulmonary Congestion.
- Atheromatous aorta.
EXAM: ELECTROCARDIOGRAM REPORT DATE: OCTOBER 25,
2021
MEASUREMENT
RATE-ATRIAL - bpm PR: - sec. QRS: 0.08 sec AXIS: -50 deg.
INTERPRETATION:
‒ Atrial Fibrillation in controlled ventricular response
Predisposing Factor:
• Family History Precipitating Factor:
• Age: 46 • Smoking
• Race • Obesity
• Sedentary lifestyle
• Decreased
Total occlusion of coronary artery
electrical
conduction
• Cardiac
dysrhythmia
(Atrial Decreased blood supply • Blood
Fibrillation) (Hypoxia) pressure of
• Elevated 105/80
troponin mmHg
level • Heart rate:
(3668.70 Myocardial Ischemia 92 BPM
ng/L) • Respiratory
rate: 28 cpm
• Sudden, MYOCARDIAL
crushing
substernal INFARCTION
chest pain
• Difficulty of
breathing
• Nausea
PHYSIOLOGY OF THE HEART
The heart weighs between 7 and 15 ounces (200 to 425 grams) and is a little larger than
the size of the fist. By the end of a long life, a person’s heart may have beat (expanded and
contracted) more than 3.5 billion times. In fact, each day, the average heart beats 100,000 times,
Location
The heart is located between the lungs in the middle of the chest, behind and slightly to the left of
the breastbone (sternum). A double-layered membrane called the pericardium surrounds the heart like a
sac. The outer layer of the pericardium surrounds the roots of the heart’s major blood vessels and is
attached by ligaments to the spinal column, diaphragm, and other parts of the body. The inner layer of the
pericardium is attached to the heart muscle. A coating of fluid separates the two layers of membrane,
Arteries: carry oxygenated blood from the heart to the rest of the body. Arteries are strong and stretchy,
which helps push blood through the circulatory system. Their elastic walls help keep blood pressure
Veins: these carry deoxygenated blood back to the heart and increase in size as they get closer to the
Capillaries: they connect the smallest arteries to the smallest veins. They have very thin walls, which
allow them to exchange compounds with surrounding tissues, such as carbon dioxide, water, oxygen,
Chambers
The heart has 4 chambers. The upper chambers are called the left and right atria, and the lower
chambers are called the left and right ventricles. A wall of muscle called the septum separates the left and
right atria and the left and right ventricles. The left ventricle is the largest and strongest chamber in the
heart. The left ventricle’s chamber walls are only about a half-inch thick, but they have enough force to
push blood through the aortic valve and into the body.
● The tricuspid valve regulates blood flow between the right atrium and right ventricle.
● The pulmonary valve controls blood flow from the right ventricle into the pulmonary
left ventricle.
● The aortic valve opens the way for oxygen-rich blood to pass from the left ventricle into
Electrical impulses from the heart muscle (the myocardium) cause the heart to contract.
This electrical signal begins in the sinoatrial (SA) node, located at the top of the right atrium.
The SA node is also called the heart’s “natural pacemaker.” An electrical impulse from this
natural pacemaker travels through the muscle fibers of the atria and ventricles, causing them to
contract. Although the SA node sends electrical impulses at a certain rate, the heart rate may still
ACTUAL PROBLEM
A. Acute Pain
• Determined It helps to
pain location determine patient’s
and pain feelings,
characteristic by appropriate
interview and intervention and
monitored pain provide basis for
scale using comparison and
numerical pain evaluation of
scale therapy
- Desired
position
- Rest periods
- Use of extra
pillows for
comfort
Dependent:
• Provided To enhance
oxygen therapy myocardial
as necessary perfusion
• Administered To maintain
prescribed acceptable level of
analgesic pain and to
(Morphine) and eradicate pain
due medications
B. Ineffective Tissue Perfusion related to reduction of blood flow
Collaborative:
• Monitor Indicators of organ
laboratory data: perfusion and
ABGs, BUN, function.
creatinine, Abnormalities in
electrolytes, coagulation may
coagulation occur as a result of
studies therapeutic
measures
C. Ineffective Breathing Pattern related to interruption of oxygen supply to cardiac muscles
Dependent:
• Administrati Administration of
on of oxygen can reduce
oxygen as shortness of breath,
ordered increase oxygen in
blood.
Collaborative:
• Refer to To assist in
Respiratory respiratory care
Therapist modalities that
include oxygen
therapy and
breathing treatments
POTENTIAL NURSING CARE PLAN
A. Risk for Decreased Cardiac Output related to infarcted muscle
Assessment Diagnosis Planning Implemention Rationale Evaluation
Subjective: Risk for Decreased Short Term: Independent: Short Term:
“Masakit ang dibdib Cardiac Output After 30 mins of • Maintained To maintain trust Goal Met
ko” as verbalized by related to infarcted nursing intervention rapport with and for After 30 mins of
the patient. muscle the patient will be patient collaboration nursing
able to: intervention, the
Objective: • Manifest and • Assessed and patient was able
• (+) dyspnea express decrease recorded VS and Compensatory to manifest and
• (+) activity episodes of O2 saturation q 4 tachycardia is a express decrease
dyspnea common response episodes of
intolerance
for patients with
• PR – 92 bpm significantly low
dyspnea.
• RR - 28 cpm Long term: blood pressure to
• BP – 105/80 After 7 hours of reduce Long Term:
cardiac
mmHg nursing intervention output. Goal Partially
Initially,
• pale, cool the patient will be this compensatoryMet
clammy, and able to: response has a After 7 hours of
diaphoretic • Demonstrate nursing
favorable effect on
adequate cardiac cardiac output but
intervention the
• CXR reveals can be harmful
output as patient was able
atheromatous when it becomes
evidence by to demonstrate
aorta persistent.
normal vital adequate cardiac
signs and output as
· increase in • Assessed and For assessment evidence by
activity tolerance noted normal vital signs
and monitoring and increase in
characteristics of
heart sounds and activity tolerance.
checked for
peripheral pulses
& capillary refill
• Noted response Over exertion
to activity increases oxygen
appropriately and consumption and
promoted rest demand
while in semi
fowler’s position
• Prepared
administration of To increase O2
oxygen supply
Collaborative: To help treat and
Refer to cardiologist prevent the
progression of the
disease
1.
1. T
o
e
a
r
c
o
B. Risk for Anxiety related to threat to health
Generic Name: The antipyretic Prophylaxis of ● Hypersensitivi ● Bleeding ● Instruct the client
Aspirin effect is due to an transient ty to aspirin or ● Dyspepsia to take the
Brand Name: action on the ischemic attack other ● Epigastric medication after
Aspilet hypothalamus, and MI salicylates Distress meals and to
Classification: resulting in heat ● Bleeding ● Nausea remain in an
Antipyretics, loss by vasodilation disorders or ● Abdominal upright position for
nonopioid of peripheral blood thrombocytop Pain and 15-30 minutes after
analgesics vessels. Anti- enia Vomiting administration.
Dosage: inflammatory are ● Rash ● Advise patient to
80 mg/tab, 1 tab mediated by a ● Urticaria report tinnitus,
Route: Oral decrease in ● Anemia unusual bleeding of
Frequency: prostaglandin ● Tinnitus gums, bruising,
OD synthesis. black tarry stool, or
fever lasting longer
than 3 days.
● Instruct patient to
avoid alcohol with
this medication to
minimize possible
gastric irritation.
DRUG NAME MECHANISM OF INDICATIONS CONTRAINDICA SIDE EFFECTS NURSING
ACTION TIONS RESPONSIBILITI
ES
MEDICATIONS
- Instructed and explained to the client that the medication is important to continue
depending on the duration that the doctor ordered for the total recovery of the patient
- Taught the patient the proper dosage and the right time to take the medication
- Informed and discussed the possible side effects and reactions that these drugs might
produce and seek medical attention if symptoms arise
EXERCISE
- Instructed the client to remember to cool down at the end of your exercise by
gradually walking slower for the last 3minute of your exercise
- Encouraged the patient to exercise at the same time daily to establish a habit and
to minimize any variables that may impact your exercise (timing of meals,
medications, work schedule)
TREATMENT
OUT-PATIENT
- Instructed the client to return to their attending physician for scheduled follow up check-
up
- Advised the client to report immediately if he/she experienced untoward symptoms such
as chest pain, headache, dizziness, or vomiting
DIET
- Choose healthier fats and oils like avocado, dark chocolates, eggs, fish (e.g. Tuna)
- Aim for two to three serves of fish and seafood per week
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